Hannah Reynolds

Hannah Reynolds’ research examines the ecology and evolution of fungal genomes and metabolism. Recent work focuses on evolutionary patterns, including gene duplication, gene cluster decay, and horizontal gene transfer. The main organisms studied are animal and human pathogens, particularly the White-nose Syndrome fungus Pseudogymnoascus destructans.

Undergraduate projects may be computer-based or take place in the laboratory.

Recent publications:

Reynolds HT, Vijayakumar V, Gluck Thaler E, Korotkin H, Matheny PB. 2018. Horizontal gene cluster transfer increased hallucinogenic mushroom diversity. doi: 10.1002/evl3.42

Reynolds HT, Slot JC, Divon HH, Lysøe E, Proctor RH, Brown DW. 2017. Differential retention of gene functions in a secondary metabolite cluster. Molecular Biology and Evolution. msx145. doi: 10.1093/molbev/msx145

Reynolds HT, Barton HA, Slot JC. 2016. Phylogenomic analysis supports a recent change in nitrate assimilation in the White-nose Syndrome pathogen, Pseudogymnoascus destructans. Fungal Ecology 23: 20-29.

Reynolds HT, Ingersoll T, Barton HA. 2015. Modeling the environmental growth of Pseudogymnoascus destructans and its impact on White-Nose Syndrome disease outcome. Journal of Wildlife Disease. 51(2):318-331.

Bonito G, Reynolds HT, Hodkinson B, Nelson J, Tuskan G, Robeson M, Schadt C, Vilgalys R. 2014. Plant host and soil origin influence fungal and bacterial assemblages in the rhizosphere of woody plants. 2014. Molecular Ecology 23(13): 3356-3370.

Reynolds HT, Barton HA. 2014. Comparison of the White-nose Syndrome agent Pseudogymnoascus destructans to cave-dwelling relatives suggests reduced saprotrophic enzyme activity. PLoS ONE. DOI: 10.1371/journal.pone.0086437

Reynolds HT, Barton HA. 2013.  White-nose Syndrome: human activity in the emergence of an extirpating mycosis. Microbiology Spectrum.  1(2). doi:10.1128/microbiolspec.OH-0008-2012.